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Home » Speakers » Michael Dustin
Michael Dustin

Michael Dustin

University of Oxford

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Michael Dustin is Professor of Immunology and Director of Research at The Kennedy Institute of Rheumatology at the University of Oxford. Prior to joining the Kennedy Institute, Dustin was a faculty member at the Skirball Institute of Biomolecular Medicine at New York University from 2001-2013 and at Washington University School of Medicine from 1993-2000. Dustin received his BA in Biology from Boston University and his PhD in Cell and Developmental Biology from Harvard University.

As described in his iBioSeminar, Dustin’s lab studies the molecular events that take place at the immunological synapse. Future research will focus on developing therapies targeted to the immunological synapse to cure chronic inflammatory illnesses such as rheumatoid arthritis.

Dustin is an active participant in the immunology community; he is a member of numerous grant review committees and journal editorial boards. His research has been recognized with many awards including the 2000 Presidential Early Career Award in Science and Engineering and the 2012 DART-NYU Biotechnology Achievement Award.

Talks with this Speaker

The Immunological Synapse

Dustin introduces the immunological synapse, the molecular interaction by which T cells and antigen presenting cells communicate. (Talk recorded in July 2015)

  • Part 1: Antigen Recognition
    Part 1: Antigen Recognition
    Audience:
    • Researcher
    Duration: 36:57
  • Part 2: Signaling and Function
    Part 2: Signaling and Function
    Audience:
    • Researcher
    Duration: 30:18
  • Part 3: Extracellular Vesicles
    Part 3: Extracellular Vesicles
    Audience:
    • Researcher
    Duration: 28:17

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This material is based upon work supported by the National Science Foundation and the National Institute of General Medical Sciences under Grant No. MCB-1052331. Any opinion, finding, conclusion, or recommendation expressed in these videos are solely those of the speaker and do not necessarily represent the views of iBiology, the National Science Foundation, the National Institutes of Health, or other iBiology funders.

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